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AP Biology Unit 7 Visual Review
A topic-by-topic visual walkthrough of Natural Selection — how selection works, population genetics, Hardy–Weinberg, evidence of evolution, phylogeny, speciation, and the origin of life.
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TOPIC 7.1
Introduction to Natural Selection
DARWIN'S FOUR OBSERVATIONS
1 · Variation
Individuals in a
population differ in
their heritable traits.
from mutation & meiosis
2 · Overproduction
More offspring are
born than can
possibly survive.
resources are limited
3 · Competition
Individuals compete
for food, mates, and
space — a struggle.
"struggle for existence"
4 · Survival
Those with favorable
traits survive &
reproduce more.
"survival of the fittest"
Putting it together
If heritable variation affects survival and reproduction, then over generations the favorable traits become
MORE common in the population. This is natural selection — the mechanism of evolution Darwin proposed.
Two key points that students often miss:
• Selection acts on the individual , but populations evolve (individuals don't "choose" to change).
• Only heritable traits can be selected — acquired traits (like a scar) are not passed on.
Variation + overproduction + competition → the fittest survive and reproduce, so populations evolve .
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TOPIC 7.2
Natural Selection — Types
THREE PATTERNS OF SELECTION
Directional
favors ONE extreme
e.g., peppered moths
Stabilizing
favors the MIDDLE
e.g., human birth weight
Disruptive
favors BOTH extremes
can lead to two forms
Fitness = reproductive success
"Fitness" doesn't mean strength — it means how many
fertile offspring an individual leaves. A trait that raises
reproductive success has higher fitness.
Adaptations & selective pressure
An adaptation is a heritable trait that improves fitness in
a given environment. The environment applies the
selective pressure — predators, climate, food, disease.
Selection can push toward one extreme, the middle, or both ends — always favoring the fittest .
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TOPIC 7.3
Artificial Selection
Humans do the selecting
In artificial selection (selective breeding), HUMANS — not the environment — choose which individuals reproduce,
based on traits we find desirable. Over generations, those traits become exaggerated in the population.
Examples: dog breeds from wolves, corn from teosinte, cattle bred for milk, and hundreds of crop varieties
(broccoli, cabbage, kale, and cauliflower all descend from one wild mustard plant).
Same mechanism, different selector
The underlying process is identical to natural selection —
heritable variation + differential reproduction. Only the
source of the "pressure" differs (humans vs. nature).
Why it's powerful evidence
Darwin used artificial selection as evidence: if humans can
reshape a species in just a few generations, nature — acting
over millions of years — can produce all of life's diversity.
A caution
Breeding for one trait can reduce genetic diversity and accidentally concentrate harmful alleles — which is
why many purebred animals suffer from inherited health problems.
Artificial selection is natural selection with a human selector — proof that selection reshapes species.
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TOPIC 7.4
Population Genetics
Evolution acts on populations
An individual can't evolve — a POPULATION does, over
generations. The gene pool is all the alleles of all the
individuals in a population.
We track evolution by measuring allele frequencies.
Microevolution = change in allele frequency
If the frequency of an allele shifts from one generation to
the next, the population has evolved. That's the modern,
genetic definition of evolution.
Allele frequency = how common an allele is (as a fraction).
Five mechanisms that change allele frequencies
1 · Natural selection — favorable alleles increase
2 · Mutation — introduces brand-new alleles
3 · Gene flow — migration moves alleles between populations
4 · Genetic drift — random chance (strong in small populations)
5 · Non-random mating — mate choice skews genotypes
If NONE of these occur, allele frequencies stay
constant — the Hardy–Weinberg baseline (Topic 7.5).
Selection is the only one that reliably produces adaptation;
the others change frequencies without "improving" fitness.
Drift & gene flow are random with respect to fitness.
Evolution is a change in allele frequencies in a population's gene pool over time.
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TOPIC 7.5
Hardy–Weinberg Equilibrium
The two equations
p + q = 1
p² + 2pq + q² = 1
What each term means
p = frequency of the dominant allele
q = frequency of the recessive allele
p² = homozygous dominant · q² = homozygous recessive
2pq = heterozygous
Tip: start from q² (the recessive phenotype) to find q.
A null model: the 5 conditions for NO evolution
Hardy–Weinberg describes a population that is NOT evolving. Allele frequencies stay constant only if ALL five hold:
1 · No natural selection 2 · No mutation 3 · No gene flow (no migration)
4 · No genetic drift (very large population) 5 · Random mating
Real populations rarely meet all five — so the equations are a BASELINE. If the observed genotype frequencies
differ from the predicted p², 2pq, q², then the population is evolving, and you can investigate why.
Remember: p and q always add to 1, and the genotype frequencies always add to 1.
p² + 2pq + q² = 1 is the "no evolution" baseline — deviations from it reveal that a population is evolving.
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TOPIC 7.6
Evidence of Evolution
Fossil record
Shows change over time and
transitional forms (e.g.,
Tiktaalik, whale ancestors).
Dated by rock layers & isotopes.
Anatomy
Homologous structures: same
bones, different uses (whale
fin, bat wing, human arm).
Vestigial organs also count.
Molecular biology
Shared DNA & protein
sequences. The more similar
the sequence, the more recent
the common ancestor.
The strongest modern evidence.
Embryology
Vertebrate embryos look strikingly similar in early
development (e.g., all have pharyngeal pouches) —
hinting at shared ancestry.
Biogeography
The geographic distribution of species reflects
evolutionary history — e.g., unique marsupials in
isolated Australia; island species resembling the mainland.
Homologous vs. analogous
Homologous = shared ancestry (divergent evolution). Analogous = similar function, NO shared ancestor
(convergent evolution — e.g., bird & insect wings). Only homology shows true relatedness.
Fossils, anatomy, molecules, embryos, and biogeography all independently point to evolution .
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TOPIC 7.7
Common Ancestry
All life shares a common ancestor
Every living thing on Earth descends from a single common ancestor. The evidence is that all organisms share
fundamental features that would be very unlikely to arise independently.
The deeper two species share these features, the more recently they diverged from a common ancestor.
Similarities that arose from a shared ancestor are called homologies.
Universal genetic code
Nearly all organisms use the
SAME codons for the same
amino acids.
Shared machinery
DNA/RNA, ribosomes, ATP,
and core metabolic pathways
are common to all cells.
Conserved genes
Some genes (like those for
ribosomal RNA) are nearly
identical across all domains.
Molecular clocks
Because mutations accumulate at a roughly steady rate, the number of DNA differences between two species
estimates how long ago they diverged — used to build and date evolutionary trees.
A universal genetic code and shared cellular machinery are strong evidence that all life is related.
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TOPIC 7.8
Continuing Evolution
Evolution is happening right now
Evolution isn't just ancient history — it's ongoing and observable, especially in organisms that reproduce quickly.
Strong selective pressures can shift populations within a human lifetime. All life continues to evolve and shares
a common set of processes for genetic change.
Antibiotic resistance
A few bacteria carry a resistance allele. Antibiotics kill
the susceptible ones; the resistant survivors reproduce.
Soon the whole population is resistant — a public-health
crisis driven by natural selection.
Other real-time examples
• Pesticide resistance in insects
• Herbicide resistance in weeds
• Viruses (like influenza and HIV) evolving fast, which is
why vaccines need frequent updates
Why fast reproducers evolve fast
Short generation times and huge population sizes mean many mutations and many rounds of selection per year.
That's why bacteria and viruses can adapt to our drugs so quickly — making resistance a moving target.
Evolution is ongoing — antibiotic & pesticide resistance are natural selection we can watch in real time.
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TOPIC 7.9
Phylogeny
A PHYLOGENETIC TREE (CLADOGRAM)
species A
species B
species C
species D
common ancestor
each node = a shared ancestor · branch = lineage
Reading a tree
Each branch point (node) represents the most recent
common ancestor of everything above it. The closer two
tips branch, the more closely related they are.
Shared derived characters
Trees are built from traits shared by a group because
they inherited it from a common ancestor. A trait that
appears at a node is shared by all descendants above it.
Key cautions
The tips are all living today — none evolved "from" another.
You can rotate branches at a node without changing the
relationships. Molecular data now guides most trees.
A phylogenetic tree maps evolutionary relationships — each node is a common ancestor .
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TOPIC 7.10
Speciation
Speciation = the formation of a new species
The biological species concept defines a species as a group that can interbreed and produce fertile offspring.
Speciation happens when populations become REPRODUCTIVELY ISOLATED and their gene pools diverge.
Allopatric speciation
A physical barrier (river, mountain, ocean) splits a
population geographically. The isolated groups adapt to
different conditions and diverge until they can no longer
interbreed. ("allo" = other place)
The most common route — e.g., Galápagos finches.
Sympatric speciation
New species form WITHOUT geographic separation —
while living in the same area. Causes include polyploidy
(common in plants), differing habitats, or mate
preferences. ("sym" = same place)
Less common; a single generation can suffice in plants.
Reproductive isolation barriers
Prezygotic barriers prevent mating or fertilization (habitat, timing, behavior, mechanical, gametic).
Postzygotic barriers act after a hybrid forms (hybrid inviability or sterility, e.g., a mule). Both keep gene pools separate.
New species arise when populations become reproductively isolated — allopatric (barrier) or sympatric (same area).
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TOPIC 7.11
Variation in Populations
Diversity = resilience
Populations with MORE genetic variation are better able to survive environmental change, disease, and new
predators — there's a higher chance some individuals carry a helpful allele. Low-diversity populations are
fragile: a single disease can wipe them out (e.g., genetically uniform crops).
Genetic drift
Random change in allele
frequencies from chance
events. Has a MUCH bigger
effect in small populations.
Bottleneck & founder
Bottleneck : a disaster slashes
a population, shrinking diversity.
Founder : a few individuals
start a new, less-diverse colony.
Gene flow
Migration moves alleles
between populations. It ADDS
variation and makes populations
more genetically similar.
Where variation comes from
The original source of all new alleles is mutation . Sexual reproduction then shuffles those alleles into new
combinations (crossing over, independent assortment, random fertilization — Topic 5.2).
More genetic variation = more resilience ; drift, bottlenecks, and founder effects reduce it.
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TOPIC 7.12
Origins of Life on Earth
~4.6 bya Earth forms
~3.8 bya first cells (prokaryotes)
~2.7 bya photosynthesis → O₂
~2.1 bya eukaryotes (endosymbiosis)
Abiotic synthesis of monomers
The early atmosphere + energy (lightning, UV, heat) could
form simple organic molecules from inorganic ones.
The Miller–Urey experiment produced amino acids this way,
showing the building blocks of life can arise abiotically.
RNA world & protobionts
RNA likely came first: it can both store information AND
catalyze reactions (as ribozymes) — so it could self-
replicate before DNA and proteins. Membrane-bound
droplets (protobionts) formed the first cell-like units.
The hypothesized sequence
Small organic molecules → polymers → self-replicating RNA → protobionts (membrane droplets) → the first
true cells. Later, endosymbiosis gave rise to eukaryotes (Topic 2.10). These are well-supported hypotheses.
Life likely began with abiotic organic molecules and an RNA world , leading to the first cells.
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How to use the visual review
Spend 30 seconds per slide before clicking next. Look at the diagram, then ask yourself: "Could I draw this from memory and explain it?"
Use the fullscreen button () on desktop for the best experience. Use arrow keys to navigate. Tap "Show all slides" to jump around.
This is great for review the night before the exam — fast, visual, and covers everything you need to remember about Unit 7's evolution content, the single most heavily weighted unit on the exam.